Outline and evaluate psychological explanations of schizophrenia Essay

Custom Student Mr. Teacher ENG 1001-04 12 July 2016

Outline and evaluate psychological explanations of schizophrenia

?Outline and evaluate psychological explanations of schizophrenia (24 marks) Brown and Birley studied stressful life events in the role of relapse in schizophrenics. They found that 50% of people experience a stressful life event e. g. death, relationship break-up, job loss etc. in the 3 weeks prior to a schizophrenic episode. A control sample reported a low and unchanging level of stressful life events over the same period. However not all evidence supports the role of life events.

For example, Van Os et al reported that patients were not more likely to have a major stressful life event in the 3 months preceding the onset of their illness. In a prospective part of the study, those patients who had experienced a major life event went on to have a lower likelihood of relapse, further-more life events after the onset of schizophrenia may be a consequence rather than a cause of schizophrenia. Bateson et al (1956) suggested that children who frequently receive contradictory messages from their parents are more likely to develop schizophrenia e. g.

a mother hugs her child but then disapprovingly tells him off for being “clingy”. This is known as the double-bind theory. There is evidence to support the double-bind theory. Berger found that schizophrenics reported a higher recall of double-bind statements by their mothers than non-schizophrenics. However this evidence might not be reliable, as patients recall may be affected by their schizophrenic. Other studies are less supportive. Liem measured patterns of parental communications in families of schizophrenic children and found no difference when compared with normal children.

Expressed emotion (EE) involves high levels of negative emotion (e. g. criticism) or high levels of positive emotion (e. g. over-protectiveness). Linszen at al (1997) found a patient returning to a family with high EE is four times more likely to relapse than a patient returning to a family with low EE. There is an issue as the whether EE is a cause or an effect of schizophrenia. Hogarty et al found that such therapy can significantly reduce intervention was the key element of the therapy or whether other aspects of family intervention may have helped.

Cognitive psychologists think that disturbed thinking processes are the cause of schizophrenia. It is thought that the mechanisms that operate in normal brains filter and process information are defective in the brains of people with schizophrenia. People with schizophrenia have no filter so they let in irrelevant information, which they are unable to interpret correctly. Studies have shown that people with schizophrenia are poor at laboratory tasks which require paying attention to only some stimuli.

However this theory doesn’t really explain the causes of schizophrenia more than just the symptoms of the disorder. Hemsley (1993) suggested that the central deflect in schizophrenia is the breakdown in the relationship between already stored memories and new incoming information. People with schizophrenic cannot activate their schemas so are subjective to sensory overload and do not know which aspects of a situation to attend and which to ignore. There has been promising research with animals offering support for Helmsley’s ideas.

Frith (1992) suggested that people with schizophrenia are unable to distinguish between actions that are caused by external forces and those happening internally. He believes that people with schizophrenic have the inability to generate willed action, the inability to monitor willed action and the inability to monitor the intentions of others. Frith suggests that these processes are part of ‘meta representation’ that allows us be aware of our goals and understand the belief of others. Friths theory has provided a comprehensive framework for explaining the symptoms of schizophrenia. However, the theory is still regarded as speculative.

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  • University/College: University of Arkansas System

  • Type of paper: Thesis/Dissertation Chapter

  • Date: 12 July 2016

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